GINA PAPADOPOULOU
POSTDOCTORAL FELLOW
My research aims to understand how homeostatic immune responses can give rise to local and distal organ inflammation, with a particular focus on neuro-immune crosstalk. During my PhD studies, I investigated how cell signaling, metabolic, and epigenetic programs regulate Th17 cell pathogenicity during central nervous system autoimmunity using mouse models of multiple sclerosis. By joining the Villablanca lab, I aim to investigate the metabolic cues that shape the education and functional programming of IL-17-producing T cells in the gut, and to determine how these processes influence their interactions with the nervous system by leveraging gut-specific conditional knockout and cell-tracing mouse models.
Villablanca Lab | KI 